In patients with the following finding, what syndrome should you suspect?
Anonymous Quiz
18%
Sturge-Weber syndrome
21%
Neurofibromatosis type I
36%
Blue rubber bleb nevus syndrome
10%
Ataxia telangiectasia
15%
Friedrichβs ataxia
π§ Blue Rubber Bleb Nevus Syndrome
π BRBNS is also known as Bean syndrome. It is a rare syndrome of venous malformations that arise in the skin and gastrointestinal tract.
π Patients present with multiple venous malformations in various organ systems including the liver, spleen, heart, eye, and central nervous system.
π Patients with BRBNS are at increased risk for gastrointestinal hemorrhage and severe iron deficiency anemia. They require a multifaceted medical management approach with hematology, dermatology, gastroenterology, and other specialties involved in their care
π Treatment is largely supportive and involves managing potential complications such as volvulus, intussusception, infarction, and gastrointestinal bleeding.
πSource:
- Blue Rubber Bleb Nevus Syndrome, StatPearls
#MCQ #Neurology
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π BRBNS is also known as Bean syndrome. It is a rare syndrome of venous malformations that arise in the skin and gastrointestinal tract.
π Patients present with multiple venous malformations in various organ systems including the liver, spleen, heart, eye, and central nervous system.
π Patients with BRBNS are at increased risk for gastrointestinal hemorrhage and severe iron deficiency anemia. They require a multifaceted medical management approach with hematology, dermatology, gastroenterology, and other specialties involved in their care
π Treatment is largely supportive and involves managing potential complications such as volvulus, intussusception, infarction, and gastrointestinal bleeding.
πSource:
- Blue Rubber Bleb Nevus Syndrome, StatPearls
#MCQ #Neurology
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π8th Annual Neuroscience Innovation Forum (NIF)
π 12 January, 2025
π San Francisco, USA
πKey features:
π·οΈFocus on Neuroscience Developments: The forum will concentrate on the latest advancements in neuroscience, covering therapeutic innovations, neurotechnology, and diagnostic tools.
πΆοΈHigh-Level Discussions: Attendees can expect over eight hours of keynotes and panel discussions led by industry leaders, addressing critical topics in the field.
More info
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π 12 January, 2025
π San Francisco, USA
πKey features:
π·οΈFocus on Neuroscience Developments: The forum will concentrate on the latest advancements in neuroscience, covering therapeutic innovations, neurotechnology, and diagnostic tools.
πΆοΈHigh-Level Discussions: Attendees can expect over eight hours of keynotes and panel discussions led by industry leaders, addressing critical topics in the field.
More info
#event
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πANT Neuromeeting Berlin 2025
π January 16-18, 2025
π Berlin, Germany
πKey features:
π·οΈScientific Presentations and Workshops: The event will feature presentations by renowned experts, hands-on showcases, workshops, and demonstrations of innovative neurotechnology.
πΆοΈNetworking Opportunities: Attendees will have the chance to connect with international peers, researchers, clinicians, and industry leaders, fostering collaboration and knowledge exchange.
More info
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π January 16-18, 2025
π Berlin, Germany
πKey features:
π·οΈScientific Presentations and Workshops: The event will feature presentations by renowned experts, hands-on showcases, workshops, and demonstrations of innovative neurotechnology.
πΆοΈNetworking Opportunities: Attendees will have the chance to connect with international peers, researchers, clinicians, and industry leaders, fostering collaboration and knowledge exchange.
More info
#event
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This lesion is associated with (the)...
Anonymous Quiz
11%
Amyotrophic lateral sclerosis
44%
Friedreichβs ataxia
11%
Multiple sclerosis
17%
Radiation myelopathy
17%
Subacute combined degeneration
π§ Friedreichβs ataxia
π Friedreichβs ataxia (FRDA) is an autosomal recessive spinocerebellar ataxia.
π In the majority of cases the disease is caused by a homozygous GAA triplet repeat expansion in the frataxin (FXN) gene, and the shorter repeat expansion length correlates with age at onset and disease severity.
π The clinical phenotype is broad, but consistently involves gait and limb ataxia, dysarthria and loss of lower limb reflexes.
π Our understanding of the genetic aetiology, pathophysiology and clinical phenotype has progressed significantly.
πSource:
- Neurosurgery Board Review, Third Edition
- Friedreichβs ataxia: clinical features, pathogenesis and management
#MCQ #Neuroradiology
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π Friedreichβs ataxia (FRDA) is an autosomal recessive spinocerebellar ataxia.
π In the majority of cases the disease is caused by a homozygous GAA triplet repeat expansion in the frataxin (FXN) gene, and the shorter repeat expansion length correlates with age at onset and disease severity.
π The clinical phenotype is broad, but consistently involves gait and limb ataxia, dysarthria and loss of lower limb reflexes.
π Our understanding of the genetic aetiology, pathophysiology and clinical phenotype has progressed significantly.
πSource:
- Neurosurgery Board Review, Third Edition
- Friedreichβs ataxia: clinical features, pathogenesis and management
#MCQ #Neuroradiology
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π Join us in BRAINet
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π7th International Virtual Conference on Neuroscience, Brain Disorders & Therapeutics
π January 25-26, 2025
π online
πKey features:
π·οΈFocus on Advancements and Challenges: The conference will explore recent treatment techniques and novel approaches in neuroscience and brain disorders, providing a platform for discussing advancements and addressing challenges in the field.
πΆοΈDiverse Participation: It aims to bring together neuroscientists, neurobiologists, neurological surgeons, neurologists, neuropathologists, neurophysiologists, neuropsychologists, and psychiatrists from various countries to share their ideas and views.
More info
#event
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π January 25-26, 2025
π online
πKey features:
π·οΈFocus on Advancements and Challenges: The conference will explore recent treatment techniques and novel approaches in neuroscience and brain disorders, providing a platform for discussing advancements and addressing challenges in the field.
πΆοΈDiverse Participation: It aims to bring together neuroscientists, neurobiologists, neurological surgeons, neurologists, neuropathologists, neurophysiologists, neuropsychologists, and psychiatrists from various countries to share their ideas and views.
More info
#event
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πKeystone Symposia: Neural Influence on Cancer, Tumor Microenvironment, and Cancer Immunology
π January 18β21, 2025
π Alberta, Canada
πKey features:
π·οΈFocus Areas:
- Exploring how nerves contribute to cancer initiation, growth, and metastasis.
- Investigating neuro-immune interactions that shape the tumor microenvironment.
- Evaluating current neuron-targeted therapeutics that could be repurposed as adjuvant cancer therapies.
More info
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π January 18β21, 2025
π Alberta, Canada
πKey features:
π·οΈFocus Areas:
- Exploring how nerves contribute to cancer initiation, growth, and metastasis.
- Investigating neuro-immune interactions that shape the tumor microenvironment.
- Evaluating current neuron-targeted therapeutics that could be repurposed as adjuvant cancer therapies.
More info
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πJanuary 2025 fMRI Speaker Series
π January 21, 2025
π Ann Arbor, USA
πKey features:
π·οΈThis event is part of the University of Michigan's Functional MRI Laboratory Speaker Series, which provides opportunities for researchers and students to learn about advancements in functional MRI and related fields.
More info
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π January 21, 2025
π Ann Arbor, USA
πKey features:
π·οΈThis event is part of the University of Michigan's Functional MRI Laboratory Speaker Series, which provides opportunities for researchers and students to learn about advancements in functional MRI and related fields.
More info
#event
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πBig Ten Neuroscience Seminar Series
π January 27, 2025
π Virtual Event
πKey features:
π·οΈShowcases neuroscience research from postdoctoral fellows and early career faculty.
πΆοΈProvides a platform for interdisciplinary collaboration and knowledge exchange.
More info
#event
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π January 27, 2025
π Virtual Event
πKey features:
π·οΈShowcases neuroscience research from postdoctoral fellows and early career faculty.
πΆοΈProvides a platform for interdisciplinary collaboration and knowledge exchange.
More info
#event
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The most appropriate treatment for a patient with multiple ischemic events and the accompanying angiogram is?
Anonymous Quiz
35%
Carotid endarterectomy
5%
Encephalomyosynangiosis
30%
Heparinization
25%
Superficial temporal artery to middle cerebral artery bypass
5%
No treatment
π―Prof. Vinko V. Dolenc
(1940-2025)
βͺοΈBorn on June 29, 1940, in SestrΕΎe near Ptuj, Slovenia.
β«οΈDedicated his life to advancing the field of neurosurgery, particularly through his groundbreaking work in microsurgical techniques.
βͺοΈAmong the first surgeons worldwide to perform ruptured aneurism surgery in the acute phase.
β«οΈHis most significant contributions came in the early 1990s with his pioneering research in parasellar space neuro-anatomy and groundbreaking surgeries on aneurysms and tumours in the cavernous sinus.
βͺοΈHis pioneering spirit, innovative approaches, and dedication to teaching have left an indelible mark on our field.
π― May his sole rest in peace.
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(1940-2025)
βͺοΈBorn on June 29, 1940, in SestrΕΎe near Ptuj, Slovenia.
β«οΈDedicated his life to advancing the field of neurosurgery, particularly through his groundbreaking work in microsurgical techniques.
βͺοΈAmong the first surgeons worldwide to perform ruptured aneurism surgery in the acute phase.
β«οΈHis most significant contributions came in the early 1990s with his pioneering research in parasellar space neuro-anatomy and groundbreaking surgeries on aneurysms and tumours in the cavernous sinus.
βͺοΈHis pioneering spirit, innovative approaches, and dedication to teaching have left an indelible mark on our field.
π― May his sole rest in peace.
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π§ Heparinization in multiple ischemic events
π Pharmacologic efforts to improve outcomes following aneurysmal subarachnoid hemorrhage (aSAH) remain disappointing, likely owing to the complex nature of post-hemorrhage brain injury.
π Previous work suggests that heparin, due to the multimodal nature of its actions, reduces the incidence of clinical vasospasm and delayed cerebral ischemia that accompany the disease.
π How heparin may mitigate the non-vasospastic pathological aspects of aSAH, particularly those related to neuroinflammation.
πSource:
- Neurosurgery Board Review, Third Edition
- Heparin and Heparin-Derivatives in Post-Subarachnoid Hemorrhage Brain Injury
#MCQ #Neuroradiology
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π Join us in BRAINet
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π Pharmacologic efforts to improve outcomes following aneurysmal subarachnoid hemorrhage (aSAH) remain disappointing, likely owing to the complex nature of post-hemorrhage brain injury.
π Previous work suggests that heparin, due to the multimodal nature of its actions, reduces the incidence of clinical vasospasm and delayed cerebral ischemia that accompany the disease.
π How heparin may mitigate the non-vasospastic pathological aspects of aSAH, particularly those related to neuroinflammation.
πSource:
- Neurosurgery Board Review, Third Edition
- Heparin and Heparin-Derivatives in Post-Subarachnoid Hemorrhage Brain Injury
#MCQ #Neuroradiology
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π Join us in BRAINet
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This postcontrast Tl-weighted MRI illustrates...
Anonymous Quiz
21%
Abscesses
17%
Gliomatosis cerebri
14%
Metastatic disease
3%
Multiple infarctus
45%
Neurofibromatosis type 2
π§ Neurofibromatosis type 2
π Neurofibromatosis type 2 is an autosomal-dominant multiple neoplasia syndrome that results from mutations in the NF2 tumour suppressor gene located on chromosome 22q.
π It has a frequency of one in 25,000 livebirths and nearly 100% penetrance by 60 years of age.
π Half of patients inherit a germline mutation from an affected parent and the remainder acquire a de novo mutation for neurofibromatosis type 2.
π Patients develop nervous system tumours (schwannomas, meningiomas, ependymomas, astrocytomas, and neurofibromas), peripheral neuropathy, ophthalmological lesions (cataracts, epiretinal membranes, and retinal hamartomas), and cutaneous lesions (skin tumours).
πSource:
- Neurosurgery Board Review, Third Edition
- Neurofibromatosis type 2: A Review study
#MCQ #Neuroradiology
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π Join us in BRAINet
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π Neurofibromatosis type 2 is an autosomal-dominant multiple neoplasia syndrome that results from mutations in the NF2 tumour suppressor gene located on chromosome 22q.
π It has a frequency of one in 25,000 livebirths and nearly 100% penetrance by 60 years of age.
π Half of patients inherit a germline mutation from an affected parent and the remainder acquire a de novo mutation for neurofibromatosis type 2.
π Patients develop nervous system tumours (schwannomas, meningiomas, ependymomas, astrocytomas, and neurofibromas), peripheral neuropathy, ophthalmological lesions (cataracts, epiretinal membranes, and retinal hamartomas), and cutaneous lesions (skin tumours).
πSource:
- Neurosurgery Board Review, Third Edition
- Neurofibromatosis type 2: A Review study
#MCQ #Neuroradiology
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π Join us in BRAINet
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#Neuroanatomy
Most of the fibers of the stria terminalis originate from the...
Most of the fibers of the stria terminalis originate from the...
Anonymous Quiz
31%
Amygdala
13%
Anterior hypothalamus
31%
Arcuate nucleus
19%
Habenula
6%
Septal nuclei
π§ Amygdala
π The amygdala is a hub of subcortical region that is crucial in a wide array of affective and motivation-related behaviors.
π While early research contributed significantly to our understanding of this region's extensive connections to other subcortical and cortical regions, recent methodological advances have enabled researchers to better understand the details of these circuits and their behavioral contributions.
π Much of this work has focused specifically on investigating the role of amygdala circuits in social cognition. In this chapter, we review both long-standing knowledge and novel research on the amygdala's structure, function, and involvement in social cognition.
πSource:
- Neurosurgery Board Review, Third Edition
- Amygdala connectivity and implications for social cognition and disorders
#MCQ #Neuroanatomy
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π Join us in BRAINet
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π The amygdala is a hub of subcortical region that is crucial in a wide array of affective and motivation-related behaviors.
π While early research contributed significantly to our understanding of this region's extensive connections to other subcortical and cortical regions, recent methodological advances have enabled researchers to better understand the details of these circuits and their behavioral contributions.
π Much of this work has focused specifically on investigating the role of amygdala circuits in social cognition. In this chapter, we review both long-standing knowledge and novel research on the amygdala's structure, function, and involvement in social cognition.
πSource:
- Neurosurgery Board Review, Third Edition
- Amygdala connectivity and implications for social cognition and disorders
#MCQ #Neuroanatomy
βββββββββββββ
π Join us in BRAINet
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